Research paperRegulation of mouse liver microsomal esterases by clofibrate and sexual hormones☆
References (42)
Carboxylesterases and amidases
- et al.
Different induction of microsomal carboxylesterases, palmitoyl-CoA hydrolase and acyl-l-carnitine hydrolase in rat liver after treatment with clofibrate
Biochem Pharmacol
(1986) - et al.
Apparent induction of microsomal-carboxylesterase activities in tissues of clofibrate-fed mice and rats
Toxicol Appl Pharmacol
(1987) - et al.
Hepatic peroxisome proliferation in rodents and its significance for humans
Food Chem Toxicol
(1993) - et al.
Peroxisome proliferators and cancer: Mechanisms and implications
Trends Pharmacol Sci
(1986) - et al.
Simultaneous purification and comparative characterization of six serine hydrolases from rat liver microsomes
Arch Biochem Biophys
(1980) - et al.
Characteristics of dehydroepiandrosterone as a peroxisome proliferator
Biochim Biophys Acta
(1991) - et al.
Sex-dependent expression and clofibrate inducibility of cytochrome P450 4A fatty acid ω-hydroxylases
J Biol Chem
(1992) - et al.
Role of testosterone in the induction of hepatic peroxisome proliferation by clofibrate
Metabolism
(1994) - et al.
Effects of testosterone, hypophysectomy and growth hormone treatment on clofibrate induction of peroxisomal β-oxidation in female rat liver
Biochem Pharmacol
(1994)
Development of surrogate substrates for juvenile hormone esterase
Arch Biochem Biophys
Substituted trifluoroketones as potent, selective inhibitors of mammalian carboxylesterases
Biochem Pharmacol
A new and rapid colorimetric determination of acetylcholinesterase activity
Biochem Pharmacol
A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
Anal Biochem
Peroxisome proliferator-activated receptors: Finding the orphan a home
Mol Cell Endocrinol
Cloning of a new member of the peroxisome proliferator-activated receptor gene family from mouse liver
J Biol Chem
Direct repeats as selective response elements for the thyroid hormone, retinoic acid, and vitamin D3 receptors
Cell
Differential regulation of soluble epoxide hydrolase by clofibrate and sexual hormones in the liver and kidneys of mice
Biochem Pharmacol
The carboxylesterases/amidases of mammalian liver and their possible significance
CRC Crit Rev Toxicol
Role of carboxylesterases in xenobiotic metabolism
Revi Biochem Toxicol
Possible physiological roles of carboxylic ester hydrolases
Drug Metab Rev
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2022, Biochemical PharmacologyCitation Excerpt :It has been reported that the CES2 promoter region contains two PPARα putative elements located at −907 and −256 [79]. Early studies have shown that di-(2-ethylhexyl)-phthalate (DHEP), a PPARα ligand, can induce hepatic CES activity in mice and rats [80,81]. Current research shows that the PPARα agonist GW7647 can also upregulate the mRNA levels of Ces1 and Ces2 in male A129/SvJ mice [82].
Hepatic carboxylesterases are differentially regulated in PPARα-null mice treated with perfluorooctanoic acid
2019, ToxicologyCitation Excerpt :One important regulator of chemical disposition in the liver is the peroxisome proliferator-activated receptor alpha (PPARα). In fact, the PPARα ligands, di-(2-ethylhexyl)-phthalate and clofibrate, have been shown to induce hepatic Ces activity in mice and rats (Hosokawa et al., 1994; Parker et al., 1996). Subsequent analysis demonstrated that the PPARα agonist GW7647 can up-regulate the mRNA levels of specific Ces subtypes, namely Ces 1d, 1e, 1f, 2c and 2e (Jones et al., 2013).
Regulation of triacylglycerol hydrolase expression by dietary fatty acids and peroxisomal proliferator-activated receptors
2003, Biochimica et Biophysica Acta - Molecular and Cell Biology of LipidsA preliminary comparison of the mouse and human genomes
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This work was supported by Grant RO1 ES02710 from NIEHS. U.C. Davis is an NIEHS Center for Environmental Health Sciences (P30 ES05707) and an EPA Center for Ecological Health Research (CR819658-010).