Brief CommunicationSlow N-acetyltransferase 2 status leads to enhanced intrastriatal dopamine depletion in 6-hydroxydopamine-lesioned rats
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Acknowledgements
Financial support from the Fritz Thyssen Stiftung für Wissenschaftsförderung, Cologne, Germany, is gratefully acknowledged. We would also like to thank Edith Sim, David Hein, Etienne Hirsch, Inke König, and in particular Andreas Hartmann for helpful discussions.
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