Research ArticleShort Communication
SULT4A1 Protects Against Oxidative-Stress Induced Mitochondrial Dysfunction in Neuronal Cells
Mohammed I. Hossain, Joshua M. Marcus, Jun Hee Lee, Patrick L. Garcia, Jean-Philippe Gagné, Guy G. Poirier, Charles N. Falany and Shaida A. Andrabi
Drug Metabolism and Disposition September 2019, 47 (9) 949-953; DOI: https://doi.org/10.1124/dmd.119.088047
Mohammed I. Hossain
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Joshua M. Marcus
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Jun Hee Lee
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Patrick L. Garcia
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Jean-Philippe Gagné
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Guy G. Poirier
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Charles N. Falany
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
Shaida A. Andrabi
Departments of Pharmacology and Toxicology (M.I.H., J.M.M., J.H.L., P.L.G., C.N.F., S.A.A.) and Neurology (S.A.A.), University of Alabama at Birmingham, Birmingham, Alabama; and Centre de recherche du CHU de Québec, Faculté de Médecine, Université Laval, Québec, Canada (J.-P.G., G.G.P.)
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Research ArticleShort Communication
SULT4A1 Preserves Mitochondrial Function
Mohammed I. Hossain, Joshua M. Marcus, Jun Hee Lee, Patrick L. Garcia, Jean-Philippe Gagné, Guy G. Poirier, Charles N. Falany and Shaida A. Andrabi
Drug Metabolism and Disposition September 1, 2019, 47 (9) 949-953; DOI: https://doi.org/10.1124/dmd.119.088047
Research ArticleShort Communication
SULT4A1 Preserves Mitochondrial Function
Mohammed I. Hossain, Joshua M. Marcus, Jun Hee Lee, Patrick L. Garcia, Jean-Philippe Gagné, Guy G. Poirier, Charles N. Falany and Shaida A. Andrabi
Drug Metabolism and Disposition September 1, 2019, 47 (9) 949-953; DOI: https://doi.org/10.1124/dmd.119.088047
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